Also in people with type external motivation (so that you look better), seek, need, require immediate solutions, and will use for this absurd and dangerous diets, supplements, creams, belts, various gadgets (To become a great investor in the “other” industry of physical exercise and health).

Reducing creams, exercises “miracle”: No such substances. In addition certain beliefs as to think that doing abdominal exercises will reduce the fat in that area have no scientific basis (Hall et al, 2005; Lopez, 2000). In respect of certain “allowances” (the melon, artichokes, dissociated, some foods do not mix with others. It is not proven scientific facts alleged benefits and harms of the same as many of these can cause to health) .

Foods or substances “miracle”: Food? all fat (not equal) except water and as said Professor Grande Covian only thing that is not fattening is what stays on the plate) and even, somehow, we could say to stop eating may not only lead to significant deficiency states and health, but lead to an opposite effect of weight loss.

About some substances of great popular acceptance as “fat burners” (mainly Carnitine), we know that carnitine is an amino acid synthesized in the liver and kidneys from lysine and methionine. The L-carnitine or butyrate (beta-hydroxy [Ganmen-N-trimethylammonium]) is an indispensable body for the penetration of long-chain fatty acids in the mitochondria of cells, which subsequently undergo oxidation. Once inside these organelles becomes carnitine acylcarnitine by the action of acyltransferase. For that fatty acids may undergo beta-oxidation need to be separated from carnitine, which collaborated with other acyltransferase. Finally, free carnitine must leave the cell, making with the help of carnitine translocase. The beta-oxidation of fatty acids released acetyl groups entering the Krebs cycle. Today we know that carnitine promotes oxidation of branched chain amino acids (Villegas, 2006).

During the year there is a redistribution of free carnitine and acylcarnitine in muscle, which does not mean you have to lose carnitine replacement. No serious study of increase in VO2 max. It enhances fatty acid oxidation in vivo, nor spare glycogen or postpone fatigue. The doses used would be 2 to 6 g / day (Villegas, 2006)
However, we know that a protein intake of high enough quality (2 g / kg / day) provides us with sufficient lysine and methionine as needed to synthesize carnitine to transport fatty acids into the mitochondria.

The more rigorous studies (Heinonen OJ.et al 1996); (Juhn MS. 2002); (Koh-Banerjee PK et al. 2005 cited by Villegas, 2006) show that:
1. Carnitine supplementation does not improve fat oxidation in vivo, or maintain glycogen stores or postpones fatigue during exercise.
2. They do not reduce body fat
3. It does not induce activation of pyruvate dehydrogenase complex (which is itself very active during exercise).
4. It does not affect VO2max
5. No carnitine deficiency demonstrated in sports efforts

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